ECO Physiotherapy Center – Health and Wellness

Why does my ankle still hurt?

Ligament injuries (sprains)

I will not only discuss the location of these injuries, but I will also try to classify them by the type of tissue involved, starting with ligaments.

A ligament, which connects bone to bone, provides passive stability to joints, and an injury to this structure is known as a sprain. Let’s start by reviewing the most common ankle injuries.

Lateral ankle sprain

A lateral ankle sprain occurs when you twist or turn your ankle inward at high speed. The ligament usually affected is the anterior talofibular ligament (ATFL) and, sometimes, the calcaneofibular ligament (CFL). Both ligaments attach to the fibula, the bone on the outside of the ankle.

Regardless of the degree of injury, proper rehabilitation is crucial because up to 40% of people develop chronic ankle instability after a first sprain.

Lateral ankle sprains are generally classified on a scale of 1 to 3. Grade 1 is a mild injury with a relatively quick return to activity. Grade 2 is a moderate injury with a slightly longer recovery time. Grade 3 is a severe injury that takes longer to rehabilitate. Generally speaking, a grade 3 injury will also present with more swelling and bruising.

This is where we get to the heart of the problem.

Chronic ankle instability

Chronic ankle instability (CAI) is defined as a “chronic ankle instability condition characterized by repetitive episodes or perceptions of the ankle giving way: ongoing symptoms such as pain, weakness, or reduced range of motion (ROM) of the ankle: Decreased self-reported function; and recurrent ankle sprains that persist for more than 1 year after the initial injury.

An early and persistent approach is essential.

Working conscientiously on the mechanoreceptors and stimulating the ankle proprioceptively is an unavoidable step for successful treatment.

General recommendations:

  1. Proper footwear: That provides support to the ankle.
  2. Orthotics: If instability is severe, it may be helpful to wear a semi-rigid orthotic when engaging in physical activity.
  3. Frequency: 2-3 sessions per week with a physical therapist, combined with daily home exercises.
  4. Follow-up: Periodic reevaluation to adjust the plan according to progress.

If the problem persists despite adequate rehabilitation, it may be necessary to consult an orthopedic surgeon to evaluate surgical options, such as ligament repair or reconstruction in severe cases.

Adductor injuries

 

An adductor injury can develop into enthesitis due to a chronic inflammatory process. Initially, an injury to the adductor muscles, such as a tear or overuse, causes microtrauma to the muscle fibers and their insertion into the bone. If not managed properly, these injuries can trigger persistent inflammation at the muscle-tendon junction (enthesis).

Factors such as overuse, inadequate rehabilitation, or lack of rest can exacerbate inflammation, promoting degenerative changes in the tissue and contributing to the development of enthesitis. This clinical picture is characterized by pain, stiffness, and tenderness in the enthesis, often associated with physical activity or direct pressure.

Enthesitis may occur more frequently in people with a genetic predisposition or systemic inflammatory diseases, such as ankylosing spondylitis, which could intensify the damage to the affected area. However, there’s good news!

With well-designed physical therapy focused on individual needs and eliminating harmful stimuli, it is possible to achieve efficient and lasting recovery. Furthermore, when combined with harnessing the body’s natural regeneration processes, balanced nutrition, and a functional and integrative approach, well-being and quality of life are further enhanced.

Bibliographic References

  1. Benjamin, M., & McGonagle, D. (2009). The enthesis organ concept and its relevance to the spondyloarthropathies. Advances in Experimental Medicine and Biology, 649, 57–70.
  2. Cook, J. L., & Purdam, C. R. (2009). Is tendon pathology a continuum? A pathology model to explain the clinical presentation of load-induced tendinopathy. British Journal of Sports Medicine, 43(6), 409–416.
  3. Rees, J. D., Stride, M., & Scott, A. (2014). Tendons–time to revisit inflammation. British Journal of Sports Medicine, 48(21), 1553–1557.
  4. Paavola, M., Kannus, P., Järvinen, T. A., Khan, K., Józsa, L., & Järvinen, M. (2002). Achilles tendinopathy. The Journal of Bone and Joint Surgery, 84(11), 2062–2076.
  5. D’Agostino, M. A., Olivieri, I., & Ferri, F. (2003). Entheseal involvement in spondylarthropathies. Clinical and Experimental Rheumatology, 21(3), 345–350.
  6. Khan, K. M., & Cook, J. L. (2000). Overuse tendon injuries: where does the pain come from? Sports Medicine, 29(3), 207–211.
  7. Biedert, R. M., & Warnke, K. (2001). Correlation between the tightness of the iliotibial band and patellofemoral pain syndrome. International Journal of Sports Medicine, 22(5), 331–336.
  8. Loppini, M., & Grassi, A. (2019). Current concepts in the management of tendon disorders. Journal of Orthopaedic Surgery and Research, 14(1), 58.
  9. Scott, A., Backman, L. J., & Speed, C. (2015). Tendinopathy: Update on Pathophysiology. Journal of Orthopaedic & Sports Physical Therapy, 45(11), 833–841.
  10. van der Heijden, R. A., Zwerver, J., & Bierma-Zeinstra, S. M. A. (2017). Tendinopathy and sports. Journal of Orthopaedic & Sports Physical Therapy, 47(5), 359–369.
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